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Nilotinib Effects on Safety, Tolerability, and Biomarkers in Alzheimer’s Disease
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Towards a biomarker system that supports disease modifying therapies in Parkinson’s disease
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Nilotinib Effects on Safety, Tolerability, and Potential Biomarkers in Parkinson Disease
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Multikinase Abl/DDR/Src Inhibition Produces Optimal Efects for Tyrosine Kinase Inhibition in Neurodegeneration
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Pharmacokinetics and pharmacodynamics of a single dose Nilotinib in individuals with Parkinson's disease
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Tau clearance improves astrocytic function and brain glutamate-glutamine cycle.
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Nilotinib Effects in Parkinson’s Disease and Dementia with Lewy Bodies
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Inhibition of DDR1-BCR signalling by nilotinib as a new therapeutic strategy for metastatic colorectal cancer
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Activating Autophagy as a Therapeutic Strategy for Parkinson's Disease.
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Discoidin domain receptor inhibition reduces neuropathology and attenuates inflammation in neurodegeneration models.
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Could cancer drugs be repurposed for use in Parkinson’s and Alzheimer’s?
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Tyrosine kinase inhibition reverses TDP-43 effects on synaptic protein expression, astrocytic function and amino acid dis-homeostasis.
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Two sides of the same coin: tyrosine kinase inhibition in cancer and neurodegeneration
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Nilotinib and bosutinib modulate pre-plaque alterations of blood immune markers and neuro-inflammation in Alzheimer's disease models.
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Tau deletion impairs intracellular β-amyloid-42 clearance and leads to more extracellular plaque deposition in gene transfer models
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Tyrosine Kinase Inhibition Regulates Early Systemic Immune Changes and Modulates the Neuroimmune Response in αSynucleinopathy
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Parkin-mediated reduction of nuclear and soluble TDP-43 reverses behavioral decline in symptomatic mice
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Nilotinib-induced autophagic changes increase endogenous parkin level and ubiquitination, leading to amyloid clearance
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Ubiquitination Increases Parkin Activity to Promote Autophagic a-Synuclein Clearance
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Tyrosine kinase inhibition facilitates autophagic SNCA/α-synuclein clearance
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Tyrosine kinase inhibition increases functional parkin‐Beclin‐1 interaction and enhances amyloid clearance and cognitive performance
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Nilotinib reverses loss of dopamine neurons and improvesmotor behavior via autophagic degradation of a-synuclein in Parkinson’s disease models
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Decreased parkin solubility is associated with impairment of autophagy in the nigrostriatum of sporadic Parkinson’s disease
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